Increased cathepsin K levels in human atherosclerotic plaques are associated with plaque instability

نویسندگان

  • Huiying Zhao
  • Xiujiao Qin
  • Shuai Wang
  • Xiwei Sun
  • Bin Dong
چکیده

[This corrects the article DOI: 10.3892/etm.2017.4935.].

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Decreased cathepsin K levels in human atherosclerotic plaques are associated with plaque instability

Investigating the determinants and dynamics of atherosclerotic plaque instability is a key area of current cardiovascular research. Extracellular matrix degradation from excessive proteolysis induced by enzymes such as cathepsin K (Cat K) is implicated in the pathogenesis of unstable plaques. The current study assessed the expression of Cat K in human unstable atherosclerotic plaques. Specimens...

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Human macrophage foam cells degrade atherosclerotic plaques through cathepsin K mediated processes

BACKGROUND Proteolytic degradation of Type I Collagen by proteases may play an important role in remodeling of atherosclerotic plaques, contributing to increased risk of plaque rupture.The aim of the current study was to investigate whether human macrophage foam cells degrade the extracellular matrix (ECM) of atherosclerotic plaques by cathepsin K mediated processes. METHODS We 1) cultured hu...

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Cathepsin K: boon or bale for atherosclerotic plaque stability?

Cathepsins, cysteine proteases of lysosomes and endosomes, play major physiological roles in general intracellular protein turnover. When they are directed outside the cell, these enzymes also take part in remodelling the extracellular matrix of bone and other tissues. Once in the extracellular matrix, the cathepsins degrade elastin and collagen. Since remodelling of the extracellular matrix of...

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Destabilizing role of cathepsin S in murine atherosclerotic plaques.

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عنوان ژورنال:

دوره 15  شماره 

صفحات  -

تاریخ انتشار 2018